| nr |
titel |
auteur |
tijdschrift |
jaar |
jaarg. |
afl. |
pagina('s) |
type |
| 1 |
A gene knockout approach in mice to identify glucose sensors controlling glucose homeostasis
|
Thorens, Bernard
|
|
2003
|
445 |
4 |
p. 482-490
|
artikel
|
| 2 |
"Back to the future…": a word from the editor
|
Rossier, B.
|
|
2003
|
445 |
4 |
p. 455
|
artikel
|
| 3 |
cAMP-dependent activation of CFTR inhibits the epithelial sodium channel (ENaC) without affecting its surface expression
|
Konstas, Angelos-Aristeidis
|
|
2003
|
445 |
4 |
p. 513-521
|
artikel
|
| 4 |
Current successes and limitations of using genetic modification for blood pressure research
|
Mullins, Linda
|
|
2003
|
445 |
4 |
p. 491-494
|
artikel
|
| 5 |
ENaC–CFTR interactions: the role of electrical coupling of ion fluxes explored in an epithelial cell model
|
Horisberger, Jean-Daniel
|
|
2003
|
445 |
4 |
p. 522-528
|
artikel
|
| 6 |
ENaC is inhibited by an increase in the intracellular Cl– concentration mediated through activation of Cl– channels
|
Kunzelmann, Karl
|
|
2003
|
445 |
4 |
p. 504-512
|
artikel
|
| 7 |
Functional interaction of CFTR and ENaC in sweat glands
|
Reddy, M.
|
|
2003
|
445 |
4 |
p. 499-503
|
artikel
|
| 8 |
Functional interaction of CFTR and ENaC in sweat glands
|
Reddy, M.
|
|
2003
|
445 |
4 |
p. 499-503
|
artikel
|
| 9 |
Inactivation of sodium-transporting proteins in the kidney
|
Rubera, I.
|
|
2003
|
445 |
4 |
p. 463-469
|
artikel
|
| 10 |
Pathophysiological role of the mineralocorticoid receptor in heart: analysis of conditional transgenic models
|
Ouvrard-Pascaud, Antoine
|
|
2003
|
445 |
4 |
p. 477-481
|
artikel
|
| 11 |
Regulation of airway surface liquid volume by human airway epithelia
|
Boucher, R.
|
|
2003
|
445 |
4 |
p. 495-498
|
artikel
|
| 12 |
The ClC-5 chloride channel knock-out mouse – an animal model for Dent's disease
|
Günther, Willy
|
|
2003
|
445 |
4 |
p. 456-462
|
artikel
|
| 13 |
Tubuloglomerular feedback in the kidney: insights from gene-targeted mice
|
Vallon, Volker
|
|
2003
|
445 |
4 |
p. 470-476
|
artikel
|
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