| no |
title |
author |
magazine |
year |
volume |
issue |
page(s) |
type |
| 1 |
Apoptin®-induced apoptosis a review
|
M. H. M. Noteborn
|
|
1999
|
4 |
5 |
p. 317-319
3 p.
|
article
|
| 2 |
Apoptin®-induced apoptosis: a review
|
Noteborn, M. H. M.
|
|
1999
|
4 |
5 |
p. 317-319
|
article
|
| 3 |
Aspirin, NSAIDs and colorectal cancerwhat do the epidemiologicalstudies show and what do they tell us about the modusoperandi?
|
K. R. Muir
|
|
1999
|
4 |
5 |
p. 389-396
8 p.
|
article
|
| 4 |
Aspirin, NSAIDs and colorectal cancer—what do the epidemiological studies show and what do they tell us about the modus operandi?
|
Muir, K. R.
|
|
1999
|
4 |
5 |
p. 389-396
|
article
|
| 5 |
Crosslinking of Fas/CD95 suppresses the CD3-mediated signalingevents in Jurkat T cells by inhibiting the association of the T-cell receptor chain with src-protein tyrosine kinases andZAP70
|
B. Kovacs
|
|
1999
|
4 |
5 |
p. 327-334
8 p.
|
article
|
| 6 |
Crosslinking of Fas/CD95 suppresses the CD3-mediated signaling events in Jurkat T cells by inhibiting the association of the T-cell receptor ζ chain with src-protein tyrosine kinases and ZAP70
|
Kovacs, B.
|
|
1999
|
4 |
5 |
p. 327-334
|
article
|
| 7 |
Fas ligand receptor or ligand?
|
M. K. Newell
|
|
1999
|
4 |
5 |
p. 311-315
5 p.
|
article
|
| 8 |
Fas ligand: receptor or ligand?
|
Newell, M. K.
|
|
1999
|
4 |
5 |
p. 311-315
|
article
|
| 9 |
Identification of an alternative form of caspase-9 in human gastriccancer cell lines a role of a caspase-9 variant in apoptosisresistance
|
M. Izawa
|
|
1999
|
4 |
5 |
p. 321-325
5 p.
|
article
|
| 10 |
Identification of an alternative form of caspase-9 in human gastric cancer cell lines: a role of a caspase-9 variant in apoptosis resistance
|
Izawa, M.
|
|
1999
|
4 |
5 |
p. 321-325
|
article
|
| 11 |
Induced apoptosis in the prevention of colorectal cancer by non-steroidal anti-inflammatory drugs
|
D. J. E. Elder
|
|
1999
|
4 |
5 |
p. 365-372
8 p.
|
article
|
| 12 |
Induced apoptosis in the prevention of colorectal cancer by non-steroidal anti-inflammatory drugs
|
Elder, D. J. E.
|
|
1999
|
4 |
5 |
p. 365-372
|
article
|
| 13 |
Janus faces of ras anti or pro-apoptotic?
|
N. Arber
|
|
1999
|
4 |
5 |
p. 383-388
6 p.
|
article
|
| 14 |
Janus faces of ras: anti or pro-apoptotic?
|
Arber, N.
|
|
1999
|
4 |
5 |
p. 383-388
|
article
|
| 15 |
Mechanism of apoptosis induced by a lysosomotropic agent, L-Leucyl-L-Leucine methyl ester
|
Takayuki Uchimoto
|
|
1999
|
4 |
5 |
p. 357-362
6 p.
|
article
|
| 16 |
Mechanism of apoptosis induced by a lysosomotropic agent, L-Leucyl-L-Leucine methyl ester
|
Uchimoto, Takayuki
|
|
1999
|
4 |
5 |
p. 357-362
|
article
|
| 17 |
Nonsteroidal anti-inflammatory drugs and the induction ofapoptosis in colon cells Evidence for PHS-dependent and PHS-independent mechanisms
|
B. Rigas
|
|
1999
|
4 |
5 |
p. 373-381
9 p.
|
article
|
| 18 |
Nonsteroidal anti-inflammatory drugs and the induction of apoptosis in colon cells: Evidence for PHS-dependent and PHS-independent mechanisms
|
Rigas, B.
|
|
1999
|
4 |
5 |
p. 373-381
|
article
|
| 19 |
NSAIDs and the GI tractpotential hazards and benefits
|
L. M. Jackson
|
|
1999
|
4 |
5 |
p. 397-402
6 p.
|
article
|
| 20 |
NSAIDs and the GI tract—potential hazards and benefits
|
Jackson, L. M.
|
|
1999
|
4 |
5 |
p. 397-402
|
article
|
| 21 |
Role of Ca2, Mg2 and K ions in determining apoptosis and extent of suppression afforded by bcl-2 during hybridoma cell culture
|
A. Ishaque
|
|
1999
|
4 |
5 |
p. 335-355
21 p.
|
article
|
| 22 |
Role of Ca2+, Mg2+ and K+ ions in determining apoptosis and extent of suppression afforded by bcl-2 during hybridoma cell culture
|
Ishaque, A.
|
|
1999
|
4 |
5 |
p. 335-355
|
article
|
Journal description