Digitale Bibliotheek
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                             10 gevonden resultaten
nr titel auteur tijdschrift jaar jaarg. afl. pagina('s) type
1 A comparative study of peritoneal mast cells from mutant IL-4 deficient and normal mice: evidence that IL-4 is not essential for mast cell development but enhances secretion via control of IgE binding and passive sensitization Banks, E.M.S.
1996
8 3 p. 190-196
7 p.
artikel
2 A fusion protein of IL-8 and a FAB antibody fragment binds to IL-8 receptors and induces neutrophil activation Hölzer, Wolfgang
1996
8 3 p. 214-221
8 p.
artikel
3 A rapid activation assay for human eosinophils based on adhesion to immobilized ICAM-1, VCAM-1 and IgG Fattah, Dilniya
1996
8 3 p. 248-259
12 p.
artikel
4 Differential changes in the concentrations of cytokine-induced neutrophil chemoattractant (CINC)-1 and CINC-2 in exudate during rat lipopolysaccharide-induced inflammation Shibata, Futoshi
1996
8 3 p. 222-226
5 p.
artikel
5 Effects of cytokines on pituitary β-endorphin and adrenal corticosterone release in vitro van der Meer, Mike J.M.
1996
8 3 p. 238-247
10 p.
artikel
6 Filgrastim (RHG-CSF) related modulation of the inflammatory response in patients at risk of sepsis or with sepsis Weiss, Manfre
1996
8 3 p. 260-265
6 p.
artikel
7 Human Cardiotrophin-1: Protein and Gene Structure, Biological and Binding Activities, and Chromosomal Localization Pennica, Diane
1996
8 3 p. 183-189
7 p.
artikel
8 Leukaemia inhibitory factor (LIF) and oncostatin M (OSM) high affinity binding require additional receptor subunits besides GP130 and GP190 Heymann, Dominique
1996
8 3 p. 197-205
9 p.
artikel
9 Subchronic glucocorticoid pretreatment reversibly attenuates IL-1β induced fever in rats; IL-6 mRNA is elevated while IL-1α and IL-1β mRNAs are suppressed, in the CNS Chai, Z.
1996
8 3 p. 227-237
11 p.
artikel
10 Type I IL-1 receptor: ligand specific conformation differences and the role of glycosylation in ligand binding Einstein, Richard
1996
8 3 p. 206-213
8 p.
artikel
                             10 gevonden resultaten
 
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