SCO-Spondin Defects and Neuroinflammation Are Conserved Mechanisms Driving Spinal Deformity across Genetic Models of Idiopathic Scoliosis
Titel:
SCO-Spondin Defects and Neuroinflammation Are Conserved Mechanisms Driving Spinal Deformity across Genetic Models of Idiopathic Scoliosis
Auteur:
Rose, Chloe D. Pompili, David Henke, Katrin Van Gennip, Jenica L.M. Meyer-Miner, Anne Rana, Rahul Gobron, Stéphane Harris, Matthew P. Nitz, Mark Ciruna, Brian