nr |
titel |
auteur |
tijdschrift |
jaar |
jaarg. |
afl. |
pagina('s) |
type |
1 |
Actin myopathy with nemaline bodies, intranuclear rods, and a heterozygous mutation in ACTA1 (Asp154Asn)
|
Schröder, J. M. |
|
2004 |
108 |
3 |
p. 250-256 |
artikel |
2 |
Biphasic NF-κB activation in the excitotoxic hippocampus
|
Caba, Ebru |
|
2004 |
108 |
3 |
p. 173-182 |
artikel |
3 |
Cortical ubiquitin-positive inclusions in frontotemporal dementia without motor neuron disease: a quantitative immunocytochemical study
|
Kövari, Enikö |
|
2004 |
108 |
3 |
p. 207-212 |
artikel |
4 |
Expression of tyrosine kinases FAK and Pyk2 in 331 human astrocytomas
|
Gutenberg, A. |
|
2004 |
108 |
3 |
p. 224-230 |
artikel |
5 |
Extracellular protein deposition correlates with glial activation and oxidative stress in Creutzfeldt-Jakob and Alzheimer’s disease
|
Everbroeck, Bart Van |
|
2004 |
108 |
3 |
p. 194-200 |
artikel |
6 |
Glyceraldehyde-derived advanced glycation end products in Alzheimer’s disease
|
Choei, Hiroshi |
|
2004 |
108 |
3 |
p. 189-193 |
artikel |
7 |
Induction of nerve growth factor mRNA in a rat dorsal root ganglion after application of a tourniquet
|
Abe, Sumiko |
|
2004 |
108 |
3 |
p. 183-188 |
artikel |
8 |
α-Internexin aggregates are abundant in neuronal intermediate filament inclusion disease (NIFID) but rare in other neurodegenerative diseases
|
Cairns, Nigel J. |
|
2004 |
108 |
3 |
p. 213-223 |
artikel |
9 |
Marked reduction of focal adhesion kinase, serum response factor and myocyte enhancer factor 2C, but increase in RhoA and myostatin in the hindlimb dy mouse muscles
|
Sakuma, Kunihiro |
|
2004 |
108 |
3 |
p. 241-249 |
artikel |
10 |
Oligodendrocytes within astrocytes (“emperipolesis”) in the white matter in Creutzfeldt-Jakob disease
|
Shintaku, Masayuki |
|
2004 |
108 |
3 |
p. 201-206 |
artikel |
11 |
Rimmed vacuoles in facioscapulohumeral muscular dystrophy: a unique ultrastructural feature
|
Neudecker, Stephan |
|
2004 |
108 |
3 |
p. 257-259 |
artikel |
12 |
Subcortical angiopathic encephalopathy in a German kindred suggests an autosomal dominant disorder distinct from CADASIL
|
Hagel, C. |
|
2004 |
108 |
3 |
p. 231-240 |
artikel |