| nr |
titel |
auteur |
tijdschrift |
jaar |
jaarg. |
afl. |
pagina('s) |
type |
| 1 |
Acetylcholinesterase-Positive Fiber Deafferentation and Cell Shrinkage in the Septohippocampal Pathway of Aged Amyloid Precursor Protein London Mutant Transgenic Mice
|
Bronfman, Francisca C.
|
|
2000
|
7 |
6 |
p. 692-
1 p.
|
artikel
|
| 2 |
Apoptosis in the Central Nervous System of Cerebral Adrenoleukodystrophy Patients
|
Feigenbaum, Valérie
|
|
2000
|
7 |
6 |
p. 600-612
13 p.
|
artikel
|
| 3 |
Author Index for Volume 7
|
|
|
2000
|
7 |
6 |
p. 697-699
3 p.
|
artikel
|
| 4 |
Binding Partners of Alzheimer's Disease Proteins: Are They Physiologically Relevant?
|
Gassen, Geert Van
|
|
2000
|
7 |
6 |
p. 692-696
5 p.
|
artikel
|
| 5 |
Brain-Derived Neurotrophic Factor in Astrocytes, Oligodendrocytes, and Microglia/Macrophages after Spinal Cord Injury
|
Dougherty, Karen D.
|
|
2000
|
7 |
6 |
p. 574-585
12 p.
|
artikel
|
| 6 |
Cellular Expression of α7 Nicotinic Acetylcholine Receptor Protein in the Temporal Cortex in Alzheimer's and Parkinson's Disease— A Stereological Approach
|
Banerjee, Carolin
|
|
2000
|
7 |
6 |
p. 666-672
7 p.
|
artikel
|
| 7 |
Costimulatory Effects of Interferon-γ and Interleukin-1β or Tumor Necrosis Factor α on the Synthesis of Aβ1-40 and Aβ1-42 by Human Astrocytes
|
Blasko, I.
|
|
2000
|
7 |
6 |
p. 682-689
8 p.
|
artikel
|
| 8 |
Enhanced Proliferation and Potassium Conductance of Schwann Cells Isolated from NF2 Schwannomas Can Be Reduced by Quinidine
|
Rosenbaum, C.
|
|
2000
|
7 |
6 |
p. 691-
1 p.
|
artikel
|
| 9 |
FAD Mutations in Presenilin-1 or Amyloid Precursor Protein Decrease the Efficacy of a γ-Secretase Inhibitor: Evidence for Direct Involvement of PS1 in the γ-Secretase Cleavage Complex
|
Xia, Weiming
|
|
2000
|
7 |
6 |
p. 673-681
9 p.
|
artikel
|
| 10 |
Homozygous Deletion in the Coding Sequence of the c-mer Gene in RCS Rats Unravels General Mechanisms of Physiological Cell Adhesion and Apoptosis
|
Nandrot, Emeline
|
|
2000
|
7 |
6 |
p. 586-599
14 p.
|
artikel
|
| 11 |
Human Cu/Zn Superoxide Dismutase (SOD1) Overexpression in Mice Causes Mitochondrial Vacuolization, Axonal Degeneration, and Premature Motoneuron Death and Accelerates Motoneuron Disease in Mice Expressing a Familial Amyotrophic Lateral Sclerosis Mutant SOD1
|
Jaarsma, Dick
|
|
2000
|
7 |
6 |
p. 623-643
21 p.
|
artikel
|
| 12 |
Hypercholesterolemia Accelerates the Alzheimer's Amyloid Pathology in a Transgenic Mouse Model
|
Refolo, Lorenzo M.
|
|
2000
|
7 |
6 |
p. 690-691
2 p.
|
artikel
|
| 13 |
Neurodegeneration Is Associated to Changes in Serum Insulin-like Growth Factors
|
Busiguina, Svetlana
|
|
2000
|
7 |
6 |
p. 657-665
9 p.
|
artikel
|
| 14 |
Neurotoxicity of the Putative Transmembrane Domain of the Prion Protein
|
Haı̈k, S.
|
|
2000
|
7 |
6 |
p. 644-656
13 p.
|
artikel
|
| 15 |
p53 Is Abnormally Elevated and Active in the CNS of Patients with Amyotrophic Lateral Sclerosis
|
Martin, Lee J.
|
|
2000
|
7 |
6 |
p. 613-622
10 p.
|
artikel
|
| 16 |
PMP22 Carrying the Trembler or Trembler-J Mutation Is Intracellularly Retained in Myelinating Schwann Cells
|
Colby, Joshua
|
|
2000
|
7 |
6 |
p. 561-573
13 p.
|
artikel
|
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