nr |
titel |
auteur |
tijdschrift |
jaar |
jaarg. |
afl. |
pagina('s) |
type |
1 |
An intriguing and hitherto unexplained co-occurrence: Depression and chronic fatigue syndrome are manifestations of shared inflammatory, oxidative and nitrosative (IO&NS) pathways
|
Maes, Michael |
|
2011 |
35 |
3 |
p. 784-794 11 p. |
artikel |
2 |
A review on the oxidative and nitrosative stress (O&NS) pathways in major depression and their possible contribution to the (neuro)degenerative processes in that illness
|
Maes, Michael |
|
2011 |
35 |
3 |
p. 676-692 17 p. |
artikel |
3 |
Beyond the serotonin hypothesis: Mitochondria, inflammation and neurodegeneration in major depression and affective spectrum disorders
|
Gardner, Ann |
|
2011 |
35 |
3 |
p. 730-743 14 p. |
artikel |
4 |
Cytokines mediated inflammation and decreased neurogenesis in animal models of depression
|
Song, Cai |
|
2011 |
35 |
3 |
p. 760-768 9 p. |
artikel |
5 |
Depression is an inflammatory disease, but cell-mediated immune activation is the key component of depression
|
Maes, Michael |
|
2011 |
35 |
3 |
p. 664-675 12 p. |
artikel |
6 |
Editorial Board
|
|
|
2011 |
35 |
3 |
p. i- 1 p. |
artikel |
7 |
Glucocorticoids, cytokines and brain abnormalities in depression
|
Zunszain, Patricia A. |
|
2011 |
35 |
3 |
p. 722-729 8 p. |
artikel |
8 |
In animal models, psychosocial stress-induced (neuro)inflammation, apoptosis and reduced neurogenesis are associated to the onset of depression
|
Kubera, Marta |
|
2011 |
35 |
3 |
p. 744-759 16 p. |
artikel |
9 |
Multiple aberrations in shared inflammatory and oxidative & nitrosative stress (IO&NS) pathways explain the co-association of depression and cardiovascular disorder (CVD), and the increased risk for CVD and due mortality in depressed patients
|
Maes, Michael |
|
2011 |
35 |
3 |
p. 769-783 15 p. |
artikel |
10 |
(Neuro)inflammation and neuroprogression as new pathways and drug targets in depression: From antioxidants to kinase inhibitors
|
Maes, Michael |
|
2011 |
35 |
3 |
p. 659-663 5 p. |
artikel |
11 |
The new ‘5-HT’ hypothesis of depression: Cell-mediated immune activation induces indoleamine 2,3-dioxygenase, which leads to lower plasma tryptophan and an increased synthesis of detrimental tryptophan catabolites (TRYCATs), both of which contribute to the onset of depression
|
Maes, M. |
|
2011 |
35 |
3 |
p. 702-721 20 p. |
artikel |
12 |
The role of zinc in neurodegenerative inflammatory pathways in depression
|
Szewczyk, Bernadeta |
|
2011 |
35 |
3 |
p. 693-701 9 p. |
artikel |